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Intestinal Failure-Associated Metabolic Bone Diseases and Response to Teriparatide

Michael Pazianas, MD^*
Charlene Compher, PhD, RD^,
Phyllis Schiavone-Gatto, MSN, CRNP
Bruce P. Kinosian, MD^*

^* University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania; and Penn Nursing and Hospital of the University of Pennsylvania Clinical Nutrition Support Service

Correspondence: Correspondence: Michael Pazianas, MD, Associate Professor of Medicine, University of Pennsylvania School of Medicine, 3615 Chestnut Street, Philadelphia, PA 19104. Electronic mail may be sent to pazianas{at}mail.med.upenn.edu.

Patients requiring home parenteral nutrition (PN) may develop metabolic bone disease, the etiology of which can be multifactorial. We report a case of significantly low bone mass in a postmenopausal woman with history of short bowel syndrome, renal impairment, and previous radiation exposure who responded to intermittent subcutaneous administration of parathyroid hormone. Her bone mineral density normalized after she completed a course of 18 months of treatment, and a bone isotope scan was negative for skeletal malignancy.

Nutrition in Clinical Practice, Vol. 21, No. 6, 605-609 (2006)
DOI: 10.1177/0115426506021006605


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